dc.contributor.author | Castro de Moura, Manuel | |
dc.contributor.author | Davalos, Veronica | |
dc.contributor.author | Planas-Serra, Laura | |
dc.contributor.author | Alvarez-Errico, Damiana | |
dc.contributor.author | Arribas, Carles | |
dc.contributor.author | Ruiz, Montserrat | |
dc.contributor.author | Aguilera-Albesa, Sergio | |
dc.contributor.author | Troya, Jesús | |
dc.contributor.author | Valencia Ramos, Juan | |
dc.contributor.author | Vélez-Santamaria, Valentina | |
dc.contributor.author | Rodríguez-Palmero, Agustí | |
dc.contributor.author | Villar-Garcia, Judit | |
dc.contributor.author | Horcajada, Juan P. | |
dc.contributor.author | Albu, Sergiu | |
dc.contributor.author | Casasnovas, Carlos | |
dc.contributor.author | Rull, Anna | |
dc.contributor.author | Reverte, Laia | |
dc.contributor.author | Dietl, Beatriz | |
dc.contributor.author | Dalmau, David | |
dc.contributor.author | Arranz, Maria J. | |
dc.contributor.author | Llucià-Carol, Laia | |
dc.contributor.author | Planas, Anna M. | |
dc.contributor.author | Pérez-Tur, Jordi | |
dc.contributor.author | Fernandez-Cadenas, Israel | |
dc.contributor.author | Villares, Paula | |
dc.contributor.author | Tenorio, Jair | |
dc.contributor.author | Colobran, Roger | |
dc.contributor.author | Martin-Nalda, Andrea | |
dc.contributor.author | Soler-Palacin, Pere | |
dc.contributor.author | Vidal, Francesc | |
dc.contributor.author | Pujol, Aurora | |
dc.contributor.author | Esteller, Manel | |
dc.date.accessioned | 2025-01-24T11:56:38Z | |
dc.date.available | 2025-01-24T11:56:38Z | |
dc.date.issued | 2021-04 | |
dc.identifier.issn | 2352-3964 | |
dc.identifier.uri | http://hdl.handle.net/10259/10038 | |
dc.description.abstract | Patients infected with the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), responsible for the coronavirus disease 2019 (COVID-19), exhibit a wide spectrum of disease behaviour. Since DNA methylation has been implicated in the regulation of viral infections and the immune system, we performed an epigenome-wide association study (EWAS) to identify candidate loci regulated by this epigenetic mark that could be involved in the onset of COVID-19 in patients without comorbidities. | es |
dc.format.mimetype | application/pdf | |
dc.language.iso | eng | es |
dc.publisher | Elsevier | es |
dc.relation.ispartof | eBioMedicine. 2021, V. 66, p. 103339 | es |
dc.rights | Attribution-NonCommercial-NoDerivatives 4.0 Internacional | * |
dc.rights.uri | http://creativecommons.org/licenses/by-nc-nd/4.0/ | * |
dc.subject | Coronavirus | es |
dc.subject | SARS-CoV-2 | es |
dc.subject | COVID-19 | es |
dc.subject | Epigenetics | es |
dc.subject | DNA methylation | es |
dc.subject.other | Medicina | es |
dc.subject.other | Medicine | es |
dc.title | Epigenome-wide association study of COVID-19 severity with respiratory failure | es |
dc.type | info:eu-repo/semantics/article | es |
dc.rights.accessRights | info:eu-repo/semantics/openAccess | es |
dc.relation.publisherversion | https://doi.org/10.1016/j.ebiom.2021.103339 | es |
dc.identifier.doi | 10.1016/j.ebiom.2021.103339 | |
dc.journal.title | eBioMedicine | es |
dc.volume.number | 66 | es |
dc.page.initial | 103339 | es |
dc.type.hasVersion | info:eu-repo/semantics/publishedVersion | es |
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